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2024

1174 Utility of immune cell receptor repertoires as predictors of development and mechanism of immune checkpoint inhibitor induced pneumonitis

Olsson-Brown, A. C., Jolly, C., Weber, D., Pan, W., Mohindra, R., Roth, A., . . . Byrne-Steele, M. (2024). 1174 Utility of immune cell receptor repertoires as predictors of development and mechanism of immune checkpoint inhibitor induced pneumonitis. In Regular and Young Investigator Award Abstracts (pp. A1302-A1303). BMJ Publishing Group Ltd. doi:10.1136/jitc-2024-sitc2024.1174

DOI
10.1136/jitc-2024-sitc2024.1174
Conference Paper

Greater mechanistic understanding of the cutaneous pathogenesis of Stevens-Johnson syndrome/toxic epidermal necrolysis can shed light on novel therapeutic strategies: a comprehensive review.

Ogiji, E. D., Aboheimed, N., Ross, K., Voller, C., Siner, R., Jensen, R. L., . . . Carr, D. F. (2024). Greater mechanistic understanding of the cutaneous pathogenesis of Stevens-Johnson syndrome/toxic epidermal necrolysis can shed light on novel therapeutic strategies: a comprehensive review.. Current opinion in allergy and clinical immunology, 24(4), 218-227. doi:10.1097/aci.0000000000000993

DOI
10.1097/aci.0000000000000993
Journal article

2023

2240P Novel genetic markers associated with immune checkpoint inhibitor induced immune-related adverse events

Chin, I. S., Mcdonald, L., Gault, A. C., Harland, M., Jolly, C., Khan, A., . . . Palles, C. (2023). 2240P Novel genetic markers associated with immune checkpoint inhibitor induced immune-related adverse events. Annals of Oncology, 34, S1154. doi:10.1016/j.annonc.2023.09.1268

DOI
10.1016/j.annonc.2023.09.1268
Journal article

TNF-α induced extracellular release of keratinocyte high-mobility group box 1 in Stevens-Johnson syndrome/toxic epidermal necrolysis: Biomarker and putative mechanism of pathogenesis

Nwikue, G., Olsson-Brown, A., Aboheimed, N., Yip, V., Jolly, C., Luchian, A., . . . Carr, D. F. (2023). TNF-α induced extracellular release of keratinocyte high-mobility group box 1 in Stevens-Johnson syndrome/toxic epidermal necrolysis: Biomarker and putative mechanism of pathogenesis. JOURNAL OF DERMATOLOGY, 50(9), 1129-1139. doi:10.1111/1346-8138.16847

DOI
10.1111/1346-8138.16847
Journal article

TNF-α-Mediated Keratinocyte Expression and Release of Matrix Metalloproteinase 9: Putative Mechanism of Pathogenesis in Stevens-Johnson Syndrome/Toxic Epidermal Necrolysis

Olsson-Brown, A., Yip, V., Ogiji, E. D., Jolly, C., Ressel, L., Sharma, A., . . . Carr, D. F. (2023). TNF-α-Mediated Keratinocyte Expression and Release of Matrix Metalloproteinase 9: Putative Mechanism of Pathogenesis in Stevens-Johnson Syndrome/Toxic Epidermal Necrolysis. JOURNAL OF INVESTIGATIVE DERMATOLOGY, 143(6), 1023-+. doi:10.1016/j.jid.2022.11.024

DOI
10.1016/j.jid.2022.11.024
Journal article

2022

The generation of HepG2 transmitochondrial cybrids to reveal the role of mitochondrial genotype in idiosyncratic drug-induced liver injury: a translational <i>in vitro</i> study

DOI
10.1101/2022.03.21.485109
Preprint

Checkpoint Inhibition Reduces the Threshold for Drug-Specific T-Cell Priming and Increases the Incidence of Sulfasalazine Hypersensitivity

Hammond, S., Olsson-Brown, A., Grice, S., Gibson, A., Gardner, J., Castrejon-Flores, J. L., . . . Naisbitt, D. J. (2022). Checkpoint Inhibition Reduces the Threshold for Drug-Specific T-Cell Priming and Increases the Incidence of Sulfasalazine Hypersensitivity. TOXICOLOGICAL SCIENCES, 186(1), 58-69. doi:10.1093/toxsci/kfab144

DOI
10.1093/toxsci/kfab144
Journal article

2021

2020

Mucosal-associated invariant T (MAIT) cells are activated in the gastrointestinal tissue of patients with combination ipilimumab and nivolumab therapy-related colitis in a pathology distinct from ulcerative colitis.

Sasson, S. C., Zaunders, J. J., Nahar, K., Munier, C. M. L., Fairfax, B. P., Olsson-Brown, A., . . . Kelleher, A. D. (2020). Mucosal-associated invariant T (MAIT) cells are activated in the gastrointestinal tissue of patients with combination ipilimumab and nivolumab therapy-related colitis in a pathology distinct from ulcerative colitis.. Clinical and experimental immunology, 202(3), 335-352. doi:10.1111/cei.13502

DOI
10.1111/cei.13502
Journal article

The utility of a differentiated preclinical liver model, HepaRG cells, in investigating delayed toxicity via inhibition of mitochondrial-replication induced by fialuridine.

Jolly, C. E., Douglas, O., Kamalian, L., Jenkins, R. E., Beckett, A. J., Penman, S. L., . . . Chadwick, A. E. (2020). The utility of a differentiated preclinical liver model, HepaRG cells, in investigating delayed toxicity via inhibition of mitochondrial-replication induced by fialuridine.. Toxicology and applied pharmacology, 403, 115163. doi:10.1016/j.taap.2020.115163

DOI
10.1016/j.taap.2020.115163
Journal article

2019

2018

Evaluating Mitotoxicity as Either a Single or Multi‐Mechanistic Insult in the Context of Hepatotoxicity

Ball, A. L., Kamalian, L., Jolly, C. E., & Chadwick, A. E. (2018). Evaluating Mitotoxicity as Either a Single or Multi‐Mechanistic Insult in the Context of Hepatotoxicity. In Unknown Book (pp. 73-92). Wiley. doi:10.1002/9781119329725.ch6

DOI
10.1002/9781119329725.ch6
Chapter