Publications
Selected publications
- Characterizing a Novel and Adjustable Noninvasive Murine Joint Loading Model (Journal article - 2011)
- Spontaneous osteoarthritis in Str/ort mice is unlikely due to greater vulnerability to mechanical trauma (Journal article - 2013)
- Intermittent applied mechanical loading induces subchondral bone thickening that may be intensified locally by contiguous articular cartilage lesions (Journal article - 2015)
- The anterior cruciate ligament in murine post-traumatic osteoarthritis: markers and mechanics (Journal article - 2022)
- Meniscal and ligament modifications in spontaneous and post-traumatic mouse models of osteoarthritis (Journal article - 2020)
- Post-traumatic osteoarthritis development is not modified by postnatal chondrocyte deletion of CCN2. (Journal article - 2020)
- Models to define the stages of articular cartilage degradation in osteoarthritis development (Journal article - 2017)
2024
Expression of semaphorin-3A in the joint and role in osteoarthritis.
Li, X., Martinez-Ramos, S., Heedge, F. T., Pitsillides, A., Bou-Gharios, G., Poulet, B., & Chenu, C. (2024). Expression of semaphorin-3A in the joint and role in osteoarthritis.. Cell Biochem Funct, 42(3), e4012. doi:10.1002/cbf.4012
2023
Characterising the relationship between muscle wasting and osteoarthritis in post-traumatic and spontaneous models of osteoarthritis
Shorter, E., Mucci, L. R., Goljanek-Whysall, K., Bou-Gharios, G., & Poulet, B. (2023). Characterising the relationship between muscle wasting and osteoarthritis in post-traumatic and spontaneous models of osteoarthritis. In INTERNATIONAL JOURNAL OF EXPERIMENTAL PATHOLOGY Vol. 104 (pp. A16-A17). Retrieved from https://www.webofscience.com/
2022
The anterior cruciate ligament in murine post-traumatic osteoarthritis: markers and mechanics
Ramos-Mucci, L., Elsheikh, A., Keenan, C., Eliasy, A., D'Aout, K., Bou-Gharios, G., . . . Poulet, B. (2022). The anterior cruciate ligament in murine post-traumatic osteoarthritis: markers and mechanics. ARTHRITIS RESEARCH & THERAPY, 24(1). doi:10.1186/s13075-022-02798-7
Identifying Novel Osteoarthritis-Associated Genes in Human Cartilage Using a Systematic Meta-Analysis and a Multi-Source Integrated Network
Shorter, E., Avelar, R., Zachariou, M., Spyrou, G. M., Raina, P., Smagul, A., . . . Poulet, B. (n.d.). Identifying Novel Osteoarthritis-Associated Genes in Human Cartilage Using a Systematic Meta-Analysis and a Multi-Source Integrated Network. International Journal of Molecular Sciences, 23(8), 4395. doi:10.3390/ijms23084395
A SYSTEMATIC META-ANALYSIS AND MULTI-SOURCE INFORMATION NETWORK IDENTIFY NOVEL OSTEOARTHRITIS-ASSOCIATED GENES
Shorter, E., Avelar, R., Spyrou, G., Zachariou, M., Raina, P., Goljanek-Whysall, K., . . . Poulet, B. (2022). A SYSTEMATIC META-ANALYSIS AND MULTI-SOURCE INFORMATION NETWORK IDENTIFY NOVEL OSTEOARTHRITIS-ASSOCIATED GENES. In OSTEOARTHRITIS AND CARTILAGE Vol. 30 (pp. S116-S117). Retrieved from https://www.webofscience.com/
BLOCKING ROR2 IMPROVES CARTILAGE INTEGRITY AND PROVIDES PAIN RELIEF IN OSTEOARTHRITIS, IN PART BY MODULATING YAP SIGNALLING
Thorup, A. -S., Wilson, J., Strachan, D., Caxaria, S., Poulet, B., Thomas, B., . . . Dell'Accio, F. (2022). BLOCKING ROR2 IMPROVES CARTILAGE INTEGRITY AND PROVIDES PAIN RELIEF IN OSTEOARTHRITIS, IN PART BY MODULATING YAP SIGNALLING. In OSTEOARTHRITIS AND CARTILAGE Vol. 30 (pp. S159). Retrieved from https://www.webofscience.com/
2021
Alterations in the gut microbiota and metabolite profiles of patients with Kashin-Beck disease, an endemic osteoarthritis in China
Wang, X., Ning, Y., Li, C., Gong, Y., Huang, R., Hu, M., . . . Guo, X. (2021). Alterations in the gut microbiota and metabolite profiles of patients with Kashin-Beck disease, an endemic osteoarthritis in China. CELL DEATH & DISEASE, 12(11). doi:10.1038/s41419-021-04322-2
BLOCKING ROR2 IMPROVES CARTILAGE INTEGRITY AND PROVIDES PAIN RELIEF IN OSTEOARTHRITIS
Thorup, A. S., Strachan, D., Caxaria, S., Poulet, B., Thomas, B., Eldridge, S., . . . Dell'accio, F. (2021). BLOCKING ROR2 IMPROVES CARTILAGE INTEGRITY AND PROVIDES PAIN RELIEF IN OSTEOARTHRITIS. In ANNALS OF THE RHEUMATIC DISEASES Vol. 80 (pp. 120-121). doi:10.1136/annrheumdis-2021-eular.2358
Comparison of the major cell populations among osteoarthritis, Kashin-Beck disease and healthy chondrocytes by single-cell RNA-seq analysis
Wang, X., Ning, Y., Zhang, P., Poulet, B., Huang, R., Gong, Y., . . . Guo, X. (2021). Comparison of the major cell populations among osteoarthritis, Kashin-Beck disease and healthy chondrocytes by single-cell RNA-seq analysis. CELL DEATH & DISEASE, 12(6). doi:10.1038/s41419-021-03832-3
Proteomics analysis of hip articular cartilage identifies differentially expressed proteins associated with osteonecrosis of the femoral head
Song, J., Wu, J., Poulet, B., Liang, J., Bai, C., Dang, X., . . . Liu, R. (2021). Proteomics analysis of hip articular cartilage identifies differentially expressed proteins associated with osteonecrosis of the femoral head. OSTEOARTHRITIS AND CARTILAGE, 29(7), 1081-1092. doi:10.1016/j.joca.2021.04.005
Syndecan-3 enhances anabolic bone formation through WNT signaling
Johnson de Sousa Brito, F. M., Butcher, A., Pisconti, A., Poulet, B., Prior, A., Charlesworth, G., . . . Daroszewska, A. (2021). Syndecan-3 enhances anabolic bone formation through WNT signaling. FASEB JOURNAL, 35(4). doi:10.1096/fj.202002024R
Cartilage-Specific Cre Recombinase Transgenes/Alleles in the Mouse
Kanakis, I., Alhashmi, M., Liu, K., Keenan, C., Mucci, L. R., Poulet, B., & Bou-Gharios, G. (2021). Cartilage-Specific Cre Recombinase Transgenes/Alleles in the Mouse. In CHONDROCYTES (Vol. 2245, pp. 23-38). doi:10.1007/978-1-0716-1119-7_3
2020
ROR2 blockade as a therapy for osteoarthritis
Thorup, A. -S., Strachan, D., Caxaria, S., Poulet, B., Thomas, B. L., Eldridge, S. E., . . . Dell'Accio, F. (2020). ROR2 blockade as a therapy for osteoarthritis. Science Translational Medicine, 12(561). doi:10.1126/scitranslmed.aax3063
The integrative analysis of DNA methylation and mRNA expression profiles confirmed the role of selenocompound metabolism pathway in Kashin-Beck disease
Li, P., Ning, Y., Wang, W., Guo, X., Poulet, B., Wang, X., . . . Xu, P. (2020). The integrative analysis of DNA methylation and mRNA expression profiles confirmed the role of selenocompound metabolism pathway in Kashin-Beck disease. CELL CYCLE, 19(18), 2351-2366. doi:10.1080/15384101.2020.1807665
Long-term bisphosphonate treatment coupled with ovariectomy in mice provokes deleterious effects on femoral neck fracture pattern and modifies tibial shape
Monzem, S., Ballester, R. Y., Javaheri, B., Poulet, B., Sonego, D. A., Pitsillides, A. A., & Souza, R. L. (2020). Long-term bisphosphonate treatment coupled with ovariectomy in mice provokes deleterious effects on femoral neck fracture pattern and modifies tibial shape. BONE & JOINT OPEN, 1(9), 512-519. doi:10.1302/2633-1462.19.BJO-2020-0117-R1
Meniscal and ligament modifications in spontaneous and post-traumatic mouse models of osteoarthritis
Ramos-Mucci, L., Javaheri, B., van ‘t Hof, R., Bou-Gharios, G., Pitsillides, A. A., Comerford, E., & Poulet, B. (n.d.). Meniscal and ligament modifications in spontaneous and post-traumatic mouse models of osteoarthritis. Arthritis Research & Therapy, 22(1). doi:10.1186/s13075-020-02261-5
Post-traumatic osteoarthritis development is not modified by postnatal chondrocyte deletion of CCN2.
Keenan, C. M., Ramos-Mucci, L., Kanakis, I., Milner, P. I., Leask, A., Abraham, D., . . . Poulet, B. (2020). Post-traumatic osteoarthritis development is not modified by postnatal chondrocyte deletion of <i>Ccn2</i>. DISEASE MODELS & MECHANISMS, 13(7). doi:10.1242/dmm.044719
Osteoarthritis-related nociceptive behaviour following mechanical joint loading correlates with cartilage damage
Heegde, F. T., Luiz, A. P., Santana-Varela, S., Magnusdottir, R., Hopkinson, M., Chang, Y., . . . Chenu, C. (2020). Osteoarthritis-related nociceptive behaviour following mechanical joint loading correlates with cartilage damage. Osteoarthritis and Cartilage, 28(3), 383-395. doi:10.1016/j.joca.2019.12.004
2019
Syndecan-3 enhances anabolic bone formation through WNT signalling
Johnson de Sousa Brito, F. M., Butcher, A., Pisconti, A., Poulet, B., Prior, A., Charlesworth, G., . . . Daroszewska, A. (2019). Syndecan-3 enhances anabolic bone formation through WNT signalling. doi:10.1101/846972
Skeletal Muscle Wasting and Its Relationship With Osteoarthritis: a Mini-Review of Mechanisms and Current Interventions
Shorter, E., Sannicandro, A. J., Poulet, B., & Goljanek-Whysall, K. (2019). Skeletal Muscle Wasting and Its Relationship With Osteoarthritis: a Mini-Review of Mechanisms and Current Interventions. CURRENT RHEUMATOLOGY REPORTS, 21(8). doi:10.1007/s11926-019-0839-4
Targeted Inhibition of Aggrecanases Prevents Articular Cartilage Degradation and Augments Bone Mass in the STR/Ort Mouse Model of Spontaneous Osteoarthritis
Kanakis, I., Liu, K., Poulet, B., Javaheri, B., van 't Hof, R. J., Pitsillides, A. A., & Bou-Gharios, G. (2019). Targeted Inhibition of Aggrecanases Prevents Articular Cartilage Degradation and Augments Bone Mass in the STR/Ort Mouse Model of Spontaneous Osteoarthritis. Arthritis and Rheumatology, 71(4), 571-582. doi:10.1002/art.40765
The role of Cryptochrome1/2 proteins in the regulation of TGF-β1 signalling pathway and their implication in tissue fibrosis
Sarumi, D., Carter, A., Fawcett, S. A., Sutton, E., Van derhorst, G. T. J., Kearns, V., . . . Pekovic-Vaughan, V. (2019). The role of Cryptochrome1/2 proteins in the regulation of TGF-β1 signalling pathway and their implication in tissue fibrosis. In INTERNATIONAL JOURNAL OF EXPERIMENTAL PATHOLOGY Vol. 100 (pp. A45). Retrieved from https://www.webofscience.com/
2018
FIBRILLIN-1 MUTANT MICE (TIGHT SKIN) SHOW INCREASED SPONTANEOUS AND TRAUMA-INDUCED OSTEOARTHRITIS DEVELOPMENT
Poulet, B. (2018). FIBRILLIN-1 MUTANT MICE (TIGHT SKIN) SHOW INCREASED SPONTANEOUS AND TRAUMA-INDUCED OSTEOARTHRITIS DEVELOPMENT. In OSTEOARTHRITIS AND CARTILAGE Vol. 26 (pp. S71). doi:10.1016/j.joca.2018.02.152
Multiple enhancer regions govern the transcription of CCN2 during embryonic development
Frost, S. L., Liu, K., Li, I. M. H., Poulet, B., Comerford, E., De Val, S., & Bou-Gharios, G. (2018). Multiple enhancer regions govern the transcription of CCN2 during embryonic development. JOURNAL OF CELL COMMUNICATION AND SIGNALING, 12(1), 231-243. doi:10.1007/s12079-017-0440-4
2017
Stable sulforaphane protects against gait anomalies and modifies bone microarchitecture in the spontaneous STR/Ort model of osteoarthritis
Javaheri, B., Poulet, B., Aljazzar, A., de Souza, R., Piles, M., Hopkinson, M., . . . Pitsillides, A. A. (2017). Stable sulforaphane protects against gait anomalies and modifies bone microarchitecture in the spontaneous STR/Ort model of osteoarthritis. BONE, 103, 308-317. doi:10.1016/j.bone.2017.07.028
Models to define the stages of articular cartilage degradation in osteoarthritis development
Poulet, B. (2017). Models to define the stages of articular cartilage degradation in osteoarthritis development. INTERNATIONAL JOURNAL OF EXPERIMENTAL PATHOLOGY, 98(3), 120-126. doi:10.1111/iep.12230
The STR/ort mouse model of spontaneous osteoarthritis - an update
Staines, K. A., Poulet, B., Wentworth, D. N., & Pitsillides, A. A. (2017). The STR/ort mouse model of spontaneous osteoarthritis - an update. OSTEOARTHRITIS AND CARTILAGE, 25(6), 802-808. doi:10.1016/j.joca.2016.12.014
THE ROLE OF MICRORNAS IN OSTEOARTHRITIS AND AGEING-RELATED FUNCTIONAL DECLINE IN JOINT TISSUE HOMEOSTASIS
McCormick, R., House, L., Poulet, B., Roebuck, M., Frostick, S., Bou-Gharious, G., & Goljanek-Whysall, K. (2017). THE ROLE OF MICRORNAS IN OSTEOARTHRITIS AND AGEING-RELATED FUNCTIONAL DECLINE IN JOINT TISSUE HOMEOSTASIS. In OSTEOARTHRITIS AND CARTILAGE Vol. 25 (pp. S290). doi:10.1016/j.joca.2017.02.487
Ageing-induced osteoarthritis in nrf2 knockout mice
Lyon, M., Pekovic-Vaughan, V., & Poulet, B. (2017). Ageing-induced osteoarthritis in nrf2 knockout mice. In INTERNATIONAL JOURNAL OF EXPERIMENTAL PATHOLOGY Vol. 98 (pp. A4). Retrieved from https://www.webofscience.com/
2016
Overexpression of TIMP-3 in chondrocytes produces transient reduction in growth plate length but permanently reduces adult bone quality and quantity
Poulet, B., Liu, K., Plumb, D., Vo, P., Shah, M., Staines, K., . . . Bou-Gharios, G. (2016). Overexpression of TIMP-3 in chondrocytes produces transient reduction in growth plate length but permanently reduces adult bone quality and quantity. PLoS ONE, 11(12). doi:10.1371/journal.pone.0167971
Chondrocyte-specific TIMP3 overexpression disrupts endochondral growth and compromises bone structure and integrity: functional impact of the chondrocyte-to-osteoblast lineage
Poulet, B., Liu, K., Plumb, D., Vo, P., Shah, M., Staines, K., . . . Carrierro, A. (2016). Chondrocyte-specific TIMP3 overexpression disrupts endochondral growth and compromises bone structure and integrity: functional impact of the chondrocyte-to-osteoblast lineage. In INTERNATIONAL JOURNAL OF EXPERIMENTAL PATHOLOGY Vol. 97 (pp. A7). Chester. doi:10.1111/iep.12211
Deficiency and also transgenic overexpression of Timp-3 both lead to compromised bone mass and architecture in vivo
Javaheri, B., Hopkinson, M., Poulet, B., Pollard, A. S., Shefelbine, S. J., Chang, Y. M., . . . Pitsillides, A. A. (2016). Deficiency and also transgenic overexpression of Timp-3 both lead to compromised bone mass and architecture in vivo. PLoS ONE, 11(8). doi:10.1371/journal.pone.0159657
Non-invasive Loading Model of Murine Osteoarthritis
Poulet, B. (2016). Non-invasive Loading Model of Murine Osteoarthritis. CURRENT RHEUMATOLOGY REPORTS, 18(7). doi:10.1007/s11926-016-0590-z
New developments in osteoarthritis and cartilage biology
Poulet, B., & Staines, K. A. (2016). New developments in osteoarthritis and cartilage biology. CURRENT OPINION IN PHARMACOLOGY, 28, 8-13. doi:10.1016/j.coph.2016.02.009
CONNECTIVE TISSUE GROWTH FACTOR DEFICIENCY IN ADULT ARTICULAR CARTILAGE PROMOTES CARTILAGE DEGENERATION AT EARLY TIMEPOINTSIN MOUSE POSTTRAUMATIC OSTEOARTHRITIS
Poulet, B., Milner, P., Kanakis, I., Carter, A., Abraham, D., Leask, A., . . . Bou-Gharios, G. (2016). CONNECTIVE TISSUE GROWTH FACTOR DEFICIENCY IN ADULT ARTICULAR CARTILAGE PROMOTES CARTILAGE DEGENERATION AT EARLY TIMEPOINTSIN MOUSE POSTTRAUMATIC OSTEOARTHRITIS. In OSTEOARTHRITIS AND CARTILAGE Vol. 24 (pp. S392). doi:10.1016/j.joca.2016.01.705
Endochondral Growth Defect and Deployment of Transient Chondrocyte Behaviors Underlie Osteoarthritis Onset in a Natural Murine Model
Staines, K. A., Madi, K., Mirczuk, S. M., Parker, S., Burleigh, A., Poulet, B., . . . Pitsillides, A. A. (2016). Endochondral Growth Defect and Deployment of Transient Chondrocyte Behaviors Underlie Osteoarthritis Onset in a Natural Murine Model. Arthritis and Rheumatology, 68(4), 880-891. doi:10.1002/art.39508
Targeting oxidative stress to reduce osteoarthritis
Poulet, B., & Beier, F. (2016). Targeting oxidative stress to reduce osteoarthritis. ARTHRITIS RESEARCH & THERAPY, 18. doi:10.1186/s13075-015-0908-7
Follistatin-like 3 deficiency modulates bone architecture and mineralisation
Poulet, B., Burleigh, A., Ptisillides, A. A., & Mukherjee, A. (2016). Follistatin-like 3 deficiency modulates bone architecture and mineralisation. In INTERNATIONAL JOURNAL OF EXPERIMENTAL PATHOLOGY Vol. 97 (pp. A14). Retrieved from https://www.webofscience.com/
2015
A homeostatic function of CXCR2 signalling in articular cartilage
Sherwood, J., Bertrand, J., Nalesso, G., Poulet, B., Pitsillides, A., Brandolini, L., . . . Dell'Accio, F. (2015). A homeostatic function of CXCR2 signalling in articular cartilage. ANNALS OF THE RHEUMATIC DISEASES, 74(12), 2207-2215. doi:10.1136/annrheumdis-2014-205546
Intermittent applied mechanical loading induces subchondral bone thickening that may be intensified locally by contiguous articular cartilage lesions
Poulet, B., de Souza, R., Kent, A. V., Saxon, L., Barker, O., Wilson, A., . . . Pitsillides, A. A. (2015). Intermittent applied mechanical loading induces subchondral bone thickening that may be intensified locally by contiguous articular cartilage lesions. Osteoarthritis and Cartilage, 23(6), 940-948. doi:10.1016/j.joca.2015.01.012
GENDER DIFFERENCES OA PROGRESSION AND THEIR LONGITUDINAL GAIT CHANGES
Piles, M., de Souza, R., Poulet, B., Pitsillides, A., & Chang, Y. -M. (2015). GENDER DIFFERENCES OA PROGRESSION AND THEIR LONGITUDINAL GAIT CHANGES. In OSTEOARTHRITIS AND CARTILAGE Vol. 23 (pp. A128). doi:10.1016/j.joca.2015.02.854
SYSTEMATIZING MURINE MENISCAL AND LIGAMENT MODIFICATIONS DURING OSTEOARTHRITIS
Poulet, B., Bou-Gharios, G., & Pitsillides, A. A. (2015). SYSTEMATIZING MURINE MENISCAL AND LIGAMENT MODIFICATIONS DURING OSTEOARTHRITIS. In OSTEOARTHRITIS AND CARTILAGE Vol. 23 (pp. A283). doi:10.1016/j.joca.2015.02.516
Fibrillin-1 is expressed in cartilage and mutation leads to OA development
Ramanayake, W., Jones, H., Orriss, I., Arnett, T., Pitsillides, A. A., Denton, C., . . . Poulet, B. (2015). Fibrillin-1 is expressed in cartilage and mutation leads to OA development. In INTERNATIONAL JOURNAL OF EXPERIMENTAL PATHOLOGY Vol. 96 (pp. A4-A5). Retrieved from https://www.webofscience.com/
2014
FIBRILLIN-1 EXPRESSION, WHICH REGULATES OF TGF-B BIOAVAILABILITY, IS MODIFIED DURING OSTEOARTHRITIS AND MUTATIONS LEAD TO OSTEOARTHRITIS
Ramanayake, W., Jones, H., Orriss, I., Arnett, T., Pitsillides, A., Denton, C., . . . Poulet, B. (2014). FIBRILLIN-1 EXPRESSION, WHICH REGULATES OF TGF-B BIOAVAILABILITY, IS MODIFIED DURING OSTEOARTHRITIS AND MUTATIONS LEAD TO OSTEOARTHRITIS. In OSTEOARTHRITIS AND CARTILAGE Vol. 22 (pp. S141). doi:10.1016/j.joca.2014.02.261
FSTL3: A NOVEL REGULATOR OF GROWTH PLATE DYNAMICS?
Burleigh, A., Staines, K. A., Poulet, B., Pitsillides, A. A., & Mukherjee, A. (2014). FSTL3: A NOVEL REGULATOR OF GROWTH PLATE DYNAMICS?. In OSTEOARTHRITIS AND CARTILAGE Vol. 22 (pp. S347). doi:10.1016/j.joca.2014.02.643
GAIT ASYMMETRY AND IMBALANCE AS POTENTIAL MARKERS OF NATURAL OSTEOARTHRITIS DEVELOPMENT IN MICE
Piles, M., Poulet, B., de Souza, R., Pitsillides, A. A., & Chang, Y. -M. (2014). GAIT ASYMMETRY AND IMBALANCE AS POTENTIAL MARKERS OF NATURAL OSTEOARTHRITIS DEVELOPMENT IN MICE. In OSTEOARTHRITIS AND CARTILAGE Vol. 22 (pp. S121). doi:10.1016/j.joca.2014.02.221
STR/ORT MICE EXHIBIT AN INHERENT ENDOCHONDRAL GROWTH DEFECT AND REDEPLOY TRANSIENT CHONDROCYTE BEHAVIOURS PRIOR TO OSTEOARTHRITIS ONSET
Staines, K. A., Parker, S., Poulet, B., Mirczuk, S., Fowkes, R. C., Hopkinson, M., . . . Pitsillides, A. A. (2014). STR/ORT MICE EXHIBIT AN INHERENT ENDOCHONDRAL GROWTH DEFECT AND REDEPLOY TRANSIENT CHONDROCYTE BEHAVIOURS PRIOR TO OSTEOARTHRITIS ONSET. In OSTEOARTHRITIS AND CARTILAGE Vol. 22 (pp. S357). doi:10.1016/j.joca.2014.02.659
FSTL3: a novel regulator of growth plate dynamics?
Burleigh, A., Staines, K., Poulet, B., Pitsillides, A. A., & Mukherjee, A. (2014). FSTL3: a novel regulator of growth plate dynamics?. In INTERNATIONAL JOURNAL OF EXPERIMENTAL PATHOLOGY Vol. 95 (pp. A6-A7). Retrieved from https://www.webofscience.com/
Follistatin-like 3 (FSTL3): A Novel Regulator of Growth Plate Dynamics
Burleigh, A., Staines, K. A., Kahlani, J. R., Poulet, B., Pitsillides, A. A., & Mukherjee, A. (2014). Follistatin-like 3 (FSTL3): A Novel Regulator of Growth Plate Dynamics. In ENDOCRINE REVIEWS Vol. 35. Retrieved from https://www.webofscience.com/
Modifications of Gait as Predictors of Natural Osteoarthritis Progression in STR/Ort Mice
Poulet, B., de Souza, R., Knights, C. B., Gentry, C., Wilson, A. M., Bevan, S., . . . Pitsillides, A. A. (2014). Modifications of Gait as Predictors of Natural Osteoarthritis Progression in STR/Ort Mice. ARTHRITIS & RHEUMATOLOGY, 66(7), 1832-1842. doi:10.1002/art.38616
Osteoarthritis and the immune phenotype of the STR/ort mouse - where do they meet?
Wentworth, D. N., Staines, K. A., Poulet, B., Constantinou, E., Palmer, D., & Pitsillides, A. A. (2014). Osteoarthritis and the immune phenotype of the STR/ort mouse - where do they meet?. In INTERNATIONAL JOURNAL OF EXPERIMENTAL PATHOLOGY Vol. 95 (pp. A36-A37). Retrieved from https://www.webofscience.com/
STR/Ort mice exhibit an endochondral growth deficit which may underpin their osteoarthritis
Staines, K. A., Parker, S., Poulet, B., Madi, K., Eastwood, D. S., Lee, P. D., & Pitsillides, A. A. (2014). STR/Ort mice exhibit an endochondral growth deficit which may underpin their osteoarthritis. In INTERNATIONAL JOURNAL OF EXPERIMENTAL PATHOLOGY Vol. 95 (pp. A30). Retrieved from https://www.webofscience.com/
The induction of both progressive and non-progressive articular cartilage lesion following trauma in C57Bl/6 mice
Poulet, B., & Pitsillides, A. A. (2014). The induction of both progressive and non-progressive articular cartilage lesion following trauma in C57Bl/6 mice. In INTERNATIONAL JOURNAL OF EXPERIMENTAL PATHOLOGY Vol. 95 (pp. A28-A29). Retrieved from https://www.webofscience.com/
2013
Npp1 is a key regulator of skeletal and soft tissue mineralisation
Hajjawi, M., MacRae, V., Huesa, C., Luis, M. J., Poulet, B., Arnett, T., & Orriss, I. (n.d.). Npp1 is a key regulator of skeletal and soft tissue mineralisation. Bone Abstracts. doi:10.1530/boneabs.01.oc6.1
Sclerostin/MEPE axis in OA: lessons from long bone development
Staines, K., Poulet, B., Farquharson, C., & Pitsillides, A. (n.d.). Sclerostin/MEPE axis in OA: lessons from long bone development. Bone Abstracts. doi:10.1530/boneabs.1.pp27
Spontaneous osteoarthritis in Str/ort mice is unlikely due to greater vulnerability to mechanical trauma
Poulet, B., Westerhof, T. A. T., Hamilton, R. W., Shefelbine, S. J., & Pitsillides, A. A. (2013). Spontaneous osteoarthritis in Str/ort mice is unlikely due to greater vulnerability to mechanical trauma. OSTEOARTHRITIS AND CARTILAGE, 21(5), 756-763. doi:10.1016/j.joca.2013.02.652
THE SCLEROSTIN AND MEPE AXIS IN THE DEVELOPMENT OF OSTEOARTHRTTIS
Staines, K. A., Poulet, B., Farquharson, C., & Pitsillides, A. A. (2013). THE SCLEROSTIN AND MEPE AXIS IN THE DEVELOPMENT OF OSTEOARTHRTTIS. In OSTEOARTHRITIS AND CARTILAGE Vol. 21 (pp. S55). doi:10.1016/j.joca.2013.02.128
THICKENING OF SUBCHONDRAL BONE IS REGIONALISED BY LOADING OF DAMAGED ARTICULAR CARTILAGE AND CONTRA-LATERAL CHANGES ARE LINKED TO GAIT MODIFICATION
Poulet, B., de Souza, R., Kent, A. V., Saxon, L., Barker, O., Wilson, A., . . . Pitsillides, A. A. (2013). THICKENING OF SUBCHONDRAL BONE IS REGIONALISED BY LOADING OF DAMAGED ARTICULAR CARTILAGE AND CONTRA-LATERAL CHANGES ARE LINKED TO GAIT MODIFICATION. In OSTEOARTHRITIS AND CARTILAGE Vol. 21 (pp. S68). doi:10.1016/j.joca.2013.02.148
Fibrillin-1 Expression and Function Is Needed For Normal Joint Function and Mutations Leads To Osteoarthritis
Ramanayake, W., Jones, H., Orriss, I., Arnett, T., Pitsillides, A., Denton, C. P., . . . Poulet, B. (2013). Fibrillin-1 Expression and Function Is Needed For Normal Joint Function and Mutations Leads To Osteoarthritis. In ARTHRITIS AND RHEUMATISM Vol. 65 (pp. S28-S29). Retrieved from https://www.webofscience.com/
Osteoarthritis severity in male offspring is linked to maternal age and parity in Str/ort mice
Poulet, B., Hesketh, F. M., Chang, Y. M., & Pitsillides, A. A. (2013). Osteoarthritis severity in male offspring is linked to maternal age and parity in Str/ort mice. In INTERNATIONAL JOURNAL OF EXPERIMENTAL PATHOLOGY Vol. 94 (pp. A14). Retrieved from https://www.webofscience.com/
Stabilising cartilage to innately protect against osteoarthritis: lessons from long bone development
Staines, K. A., Poulet, B., Farquharson, C., & Pitsillides, A. A. (2013). Stabilising cartilage to innately protect against osteoarthritis: lessons from long bone development. In INTERNATIONAL JOURNAL OF EXPERIMENTAL PATHOLOGY Vol. 94 (pp. A3-A4). Retrieved from https://www.webofscience.com/
2012
Time-series transcriptional profiling yields new perspectives on susceptibility to murine osteoarthritis
Poulet, B., Ulici, V., Stone, T. C., Pead, M., Gburcik, V., Constantinou, E., . . . Pitsillides, A. A. (2012). Time-series transcriptional profiling yields new perspectives on susceptibility to murine osteoarthritis. ARTHRITIS AND RHEUMATISM, 64(10), 3256-3266. doi:10.1002/art.34572
2011
LOAD-INDUCED PROGRESSIVE LESIONS ARE ASSOCIATED WITH PROTEOGLYCAN LOSS BUT NOT WITH CELL DEATH
Poulet, B., Khan, I. M., Archer, C. W., & Pistillides, A. A. (2011). LOAD-INDUCED PROGRESSIVE LESIONS ARE ASSOCIATED WITH PROTEOGLYCAN LOSS BUT NOT WITH CELL DEATH. In OSTEOARTHRITIS AND CARTILAGE Vol. 19 (pp. S16). doi:10.1016/S1063-4584(11)60048-4
LOAD-INDUCED SUBCHONDRAL BONE THICKENING IN MICE WITH OR WITHOUT ARTICULAR CARTILAGE LESIONS
Poulet, B., Saxon, L., Barker, O., De Souza, R., & Pitsillides, A. A. (2011). LOAD-INDUCED SUBCHONDRAL BONE THICKENING IN MICE WITH OR WITHOUT ARTICULAR CARTILAGE LESIONS. In OSTEOARTHRITIS AND CARTILAGE Vol. 19 (pp. S97). doi:10.1016/S1063-4584(11)60222-7
Localised thickening of subchondral bone in a model of loading-induced articular cartilage lesions: Product of cartilage damage or not?
Poulet, B., Saxon, L., Barker, O., de Souza, R., & Pitsillides, A. A. (2011). Localised thickening of subchondral bone in a model of loading-induced articular cartilage lesions: Product of cartilage damage or not?. In BONE Vol. 48 (pp. S100). doi:10.1016/j.bone.2011.03.150
The 70-gene expression profile, Mammaprint, for breast cancer patients in mainly European hospitals
Zanconati, F., Cusumano, P., Tinterri, C., Di Napoli, A., Holzik, M. L., Poulet, B., . . . Sapino, A. (2011). The 70-gene expression profile, Mammaprint, for breast cancer patients in mainly European hospitals. In BREAST Vol. 20 (pp. S45). doi:10.1016/S0960-9776(11)70146-1
Characterizing a Novel and Adjustable Noninvasive Murine Joint Loading Model
Poulet, B., Hamilton, R. W., Shefelbine, S., & Pitsillides, A. A. (2011). Characterizing a Novel and Adjustable Noninvasive Murine Joint Loading Model. ARTHRITIS AND RHEUMATISM, 63(1), 137-147. doi:10.1002/art.27765
Chondrocyte death alone is not sufficient to induce AC lesion progression
Poulet, B., Khan, I. M., Archer, C. W., & Pitsillides, A. A. (2011). Chondrocyte death alone is not sufficient to induce AC lesion progression. In INTERNATIONAL JOURNAL OF EXPERIMENTAL PATHOLOGY Vol. 92 (pp. A29). Retrieved from https://www.webofscience.com/
Subchondral bone thickening, with or without articular cartilage lesions, in response to joint loading
Poulet, B., Saxon, L., Barker, O., de Souza, R., & Pitsillides, A. A. (2011). Subchondral bone thickening, with or without articular cartilage lesions, in response to joint loading. In INTERNATIONAL JOURNAL OF EXPERIMENTAL PATHOLOGY Vol. 92 (pp. A28). Retrieved from https://www.webofscience.com/
2010
GENE EXPRESSION PROFILE IN THE ARTICULAR CARTILAGE OF A NATURAL MODEL OF MOUSE OSTEOARTHRITIS
Poulet, B., Ulici, V., Pead, M., Beier, F., & Pitsillides, A. A. (2010). GENE EXPRESSION PROFILE IN THE ARTICULAR CARTILAGE OF A NATURAL MODEL OF MOUSE OSTEOARTHRITIS. In OSTEOARTHRITIS AND CARTILAGE Vol. 18 (pp. S169). doi:10.1016/S1063-4584(10)60410-4
IS FOLLISTATIN-LIKE 3 OR FSTL3 NECESSARY FOR JOINT HEALTH?
Poulet, B., Mcneil, J., & Pitsillides, A. A. (2010). IS FOLLISTATIN-LIKE 3 OR FSTL3 NECESSARY FOR JOINT HEALTH?. In OSTEOARTHRITIS AND CARTILAGE Vol. 18 (pp. S13). doi:10.1016/S1063-4584(10)60037-4
MOUSE ANKLE JOINTS ARE, UNLIKE KNEES, RESISTANT TO LOAD-INDUCED LESION FORMATION
Poulet, B., Simmons, J., & Pitsillides, A. A. (2010). MOUSE ANKLE JOINTS ARE, UNLIKE KNEES, RESISTANT TO LOAD-INDUCED LESION FORMATION. In OSTEOARTHRITIS AND CARTILAGE Vol. 18 (pp. S54). doi:10.1016/S1063-4584(10)60132-X
Deletion of Follistatin-Like 3 (FSTL3) Results in Development of Osteoarthritis
Poulet, B., McNeill, J., Pitsillides, A., & Mukherjee, A. (2010). Deletion of Follistatin-Like 3 (FSTL3) Results in Development of Osteoarthritis. In ENDOCRINE REVIEWS Vol. 31 (pp. S2468). Retrieved from https://www.webofscience.com/
Follistatin-like 3 protein (FSTL3) is necessary for synovial joint integrity in aged mice
Poulet, B., Mcneil, J., Mukherjee, A., & Pitsillides, A. A. (2010). Follistatin-like 3 protein (FSTL3) is necessary for synovial joint integrity in aged mice. In INTERNATIONAL JOURNAL OF EXPERIMENTAL PATHOLOGY Vol. 91 (pp. A20). Retrieved from https://www.webofscience.com/
Mechanical loading of murine knee joints: determining the interplay between genetics and mechanical challenge in the development of OA
Poulet, B., & Pitsillides, A. A. (2010). Mechanical loading of murine knee joints: determining the interplay between genetics and mechanical challenge in the development of OA. In INTERNATIONAL JOURNAL OF EXPERIMENTAL PATHOLOGY Vol. 91 (pp. A28-A29). Retrieved from https://www.webofscience.com/
2009
005 USE OF A NOVEL IN VIVO KNEE JOINT LOADING MODEL TO DETERMINE THE INTERPLAY BETWEEN GENETIC AND MECHANICAL FACTORS IN THE INITIATION AND PROGRESSION OF MURINE OA
Poulet, B., Hamilton, R. W., Lee, P. D., Shefelbine, S. J., & Pitsillides, A. A. (2009). 005 USE OF A NOVEL IN VIVO KNEE JOINT LOADING MODEL TO DETERMINE THE INTERPLAY BETWEEN GENETIC AND MECHANICAL FACTORS IN THE INITIATION AND PROGRESSION OF MURINE OA. Osteoarthritis and Cartilage, 17, S11. doi:10.1016/s1063-4584(09)60028-5
Past, Present and Future Insights into the Understanding and Treatment of Osteoarthritis: Molecular and Mechanical Approaches
Past, Present and Future Insights into the Understanding and Treatment of Osteoarthritis: Molecular and Mechanical Approaches (2009). In International Journal of Experimental Pathology Vol. 90. Wiley. doi:10.1111/j.1365-2613.2008.00612.x