2024
Liew, F., Efstathiou, C., Fontanella, S., Richardson, M., Saunders, R., Swieboda, D., . . . ISARIC investigators. (2024). Large-scale phenotyping of patients with long COVID post-hospitalization reveals mechanistic subtypes of disease.. Nature immunology, 25(4), 607-621. doi:10.1038/s41590-024-01778-0DOI: 10.1038/s41590-024-01778-0
Thin, K. A., Cross, A., Angsuwatcharakon, P., Mutirangura, A., Puttipanyalears, C., & Edwards, S. W. (2024). Changes in immune cell subtypes during ageing.. Archives of gerontology and geriatrics, 122, 105376. doi:10.1016/j.archger.2024.105376DOI: 10.1016/j.archger.2024.105376
Lord, J. M., Veenith, T., Sullivan, J., Sharma-Oates, A., Richter, A. G., Greening, N. J., . . . ISARIC4C investigators. (2024). Accelarated immune ageing is associated with COVID-19 disease severity.. Immunity & ageing : I & A, 21(1), 6. doi:10.1186/s12979-023-00406-zDOI: 10.1186/s12979-023-00406-z
2023
Taquet, M., Skorniewska, Z., Zetterberg, H., Geddes, J. R., Mummery, C. J., Chalmers, J. D., . . . PHOSP-COVID Study Collaborative Group . (2024). Post-acute COVID-19 neuropsychiatric symptoms are not associated with ongoing nervous system injury.. Brain communications, 6(1), fcad357. doi:10.1093/braincomms/fcad357DOI: 10.1093/braincomms/fcad357
Elneima, O., McAuley, H. J. C., Leavy, O. C., Chalmers, J. D., Horsley, A., Ho, L. -P., . . . Williams, N. (2023). Cohort Profile: Post-Hospitalisation COVID-19 (PHOSP-COVID) study. International Journal of Epidemiology, 53(1). doi:10.1093/ije/dyad165DOI: 10.1093/ije/dyad165
Jackson, C., Stewart, I., Plekhanova, T., Cunningham, P. S., Hazel, A. L., Al-Sheklly, B., . . . PHOSP-COVID, S. C. G. (2023). Effects of sleep disturbance on dyspnoea and impaired lung function following hospital admission due to COVID-19 in the UK: a prospective multicentre cohort study. LANCET RESPIRATORY MEDICINE, 11(8), 673-684. doi:10.1016/S2213-2600(23)00124-8DOI: 10.1016/S2213-2600(23)00124-8
Pascall, D. J., Vink, E., Blacow, R., Bulteel, N., Campbell, A., Campbell, R., . . . COVID-19 Genomics UK (COG-UK) Consortium. (2023). The SARS-CoV-2 Alpha variant was associated with increased clinical severity of COVID-19 in Scotland: A genomics-based retrospective cohort analysis.. PloS one, 18(4), e0284187. doi:10.1371/journal.pone.0284187DOI: 10.1371/journal.pone.0284187
Moore, S., Kronsteiner, B., Longet, S., Adele, S., Deeks, A., Liu, C., . . . Consortium, P. I. T. C. H. (2023). Evolution of Long-Term Hybrid Immunity in Healthcare Workers after Different Covid-19 Vaccination Regimens: A Longitudinal Observational Cohort Study. Med. doi:10.1016/j.medj.2023.02.004DOI: 10.1016/j.medj.2023.02.004
Moore, S. C., Kronsteiner, B., Longet, S., Adele, S., Deeks, A. S., Liu, C., . . . PITCH, C. (2023). Evolution of long-term vaccine-induced and hybrid immunity in healthcare workers after different COVID-19 vaccine regimens. MED, 4(3), 191-+. doi:10.1016/j.medj.2023.02.004DOI: 10.1016/j.medj.2023.02.004
Cross, A., Hawkes, J., Frankland, H., Mediana, A., Wright, H., Goodson, N., . . . Moots, R. (2023). Neutrophil function following treatment of psoriatic arthritis patients with secukinumab: altered cytokine signalling but no impairment of host defence. Rheumatology. doi:10.1093/rheumatology/kead007DOI: 10.1093/rheumatology/kead007
Liew, F., Talwar, S., Cross, A., Willett, B. J., Scott, S., Logan, N., . . . Openshaw, P. J. M. (2023). SARS-CoV-2-specific nasal IgA wanes 9 months after hospitalisation with COVID-19 and is not induced by subsequent vaccination. EBIOMEDICINE, 87. doi:10.1016/j.ebiom.2022.104402DOI: 10.1016/j.ebiom.2022.104402
2022
Willett, B. J., Grove, J., MacLean, O. A., Wilkie, C., De Lorenzo, G., Furnon, W., . . . Thomson, E. C. (2022). SARS-CoV-2 Omicron is an immune escape variant with an altered cell entry pathway (vol 7, pg 1161, 2022). NATURE MICROBIOLOGY, 7(10), 1709. doi:10.1038/s41564-022-01241-6DOI: 10.1038/s41564-022-01241-6
Willett, B. J., Grove, J., MacLean, O. A., Wilkie, C., De Lorenzo, G., Furnon, W., . . . Thomson, E. C. (2022). SARS-CoV-2 Omicron is an immune escape variant with an altered cell entry pathway. NATURE MICROBIOLOGY, 7(8), 1161-+. doi:10.1038/s41564-022-01143-7DOI: 10.1038/s41564-022-01143-7
Smallman-Raynor, M. R., Cliff, A. D., & COVID-19 Genomics UK (COG-UK) Consortium. (2022). Spatial growth rate of emerging SARS-CoV-2 lineages in England, September 2020-December 2021.. Epidemiology and infection, 150, e145. doi:10.1017/s0950268822001285DOI: 10.1017/s0950268822001285
Nickbakhsh, S., Hughes, J., Christofidis, N., Griffiths, E., Shaaban, S., Enright, J., . . . Smith-Palmer, A. (2022). Genomic epidemiology of SARS-CoV-2 in a university outbreak setting and implications for public health planning. Scientific Reports, 12(1). doi:10.1038/s41598-022-15661-1DOI: 10.1038/s41598-022-15661-1
Chokesuwattanaskul, S., Alarcon, M. F., Mangalakumaran, S., Grosman, R., Cross, A. L., Chapman, E. A., . . . Wright, H. L. (2022). Metabolic Profiling of Rheumatoid Arthritis Neutrophils Reveals Altered Energy Metabolism That Is Not Affected by JAK Inhibition. METABOLITES, 12(7). doi:10.3390/metabo12070650DOI: 10.3390/metabo12070650
Klaser, K., Molteni, E., Graham, M., Canas, L. S., Osterdahl, M. F., Antonelli, M., . . . Duncan, E. L. (2022). COVID-19 due to the B.1.617.2 (Delta) variant compared to B.1.1.7 (Alpha) variant of SARS-CoV-2: a prospective observational cohort study. SCIENTIFIC REPORTS, 12(1). doi:10.1038/s41598-022-14016-0DOI: 10.1038/s41598-022-14016-0
Vöhringer, H. S., Sanderson, T., Sinnott, M., De Maio, N., Nguyen, T., Goater, R., . . . Gerstung, M. (2022). Publisher Correction: Genomic reconstruction of the SARS CoV-2 epidemic in England.. Nature, 606(7915), E18. doi:10.1038/s41586-022-04887-8DOI: 10.1038/s41586-022-04887-8
Wright, D. W., Harvey, W. T., Hughes, J., Cox, M., Peacock, T. P., Colquhoun, R., . . . Carabelli, A. M. (2022). Tracking SARS-CoV-2 mutations and variants through the COG-UK-Mutation Explorer.. Virus evolution, 8(1), veac023. doi:10.1093/ve/veac023DOI: 10.1093/ve/veac023
Angyal, A., Longet, S., Moore, S. C., Payne, R. P., Harding, A., Tipton, T., . . . de, S. T. I. (2022). T-cell and antibody responses to first BNT162b2 vaccine dose in previously infected and SARS-CoV-2-naive UK health-care workers: a multicentre prospective cohort study. LANCET MICROBE, 3(1), E21-E31. doi:10.1016/S2666-5247(21)00275-5DOI: 10.1016/S2666-5247(21)00275-5
2021
de, S. T. I., Liu, G., Lindsey, B. B., Dong, D., Moore, S. C., Hsu, N. S., . . . Dong, T. (2021). The impact of viral mutations on recognition by SARS-CoV-2 specific T cells. ISCIENCE, 24(11). doi:10.1016/j.isci.2021.103353DOI: 10.1016/j.isci.2021.103353
Payne, R. P., Longet, S., Austin, J. A., Skelly, D. T., Dejnirattisai, W., Adele, S., . . . Dunachie, S. (2021). Immunogenicity of standard and extended dosing intervals of BNT162b2 mRNA vaccine. CELL, 184(23), 5699-+. doi:10.1016/j.cell.2021.10.011DOI: 10.1016/j.cell.2021.10.011
Evans, R. A., McAuley, H., Harrison, E. M., Shikotra, A., Singapuri, A., Sereno, M., . . . Brightling, C. E. (2021). Physical, cognitive, and mental health impacts of COVID-19 after hospitalisation (PHOSP-COVID): a UK multicentre, prospective cohort study. LANCET RESPIRATORY MEDICINE, 9(11), 1275-1287. doi:10.1016/S2213-2600(21)00383-0DOI: 10.1016/S2213-2600(21)00383-0
Zhan, D., Cross, A., Wright, H. L., Moots, R. J., Edwards, S. W., & Honsawek, S. (2021). Internalization of Neutrophil-Derived Microvesicles Modulates TNFα-Stimulated Proinflammatory Cytokine Production in Human Fibroblast-Like Synoviocytes. INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES, 22(14). doi:10.3390/ijms22147409DOI: 10.3390/ijms22147409
T-Cell and Antibody Responses to First BNT162b2 Vaccine Dose in Previously SARS-CoV-2-Infected and Infection-Naive UK Healthcare Workers: A Multicentre, Prospective, Observational Cohort Study (Preprint)
DOI: 10.2139/ssrn.3820576
2020
Cross, A. L., Hawkes, J., Wright, H. L., Moots, R. J., & Edwards, S. W. (2020). APPA (apocynin and paeonol) modulates pathological aspects of human neutrophil function, without supressing antimicrobial ability, and inhibits TNFα expression and signalling. INFLAMMOPHARMACOLOGY, 28(5), 1223-1235. doi:10.1007/s10787-020-00715-5DOI: 10.1007/s10787-020-00715-5
SECUKINUMAB THERAPY DOES NOT AFFECT NEUTROPHIL HOST DEFENCE IN PSORIATIC ARTHRITIS (Conference Paper)
Cross, A., Hawkes, J., Frankland, H., Mediana, A., Wright, H., Goodson, N., . . . Moots, R. (2020). SECUKINUMAB THERAPY DOES NOT AFFECT NEUTROPHIL HOST DEFENCE IN PSORIATIC ARTHRITIS. In ANNALS OF THE RHEUMATIC DISEASES Vol. 79 (pp. 1353-1354). doi:10.1136/annrheumdis-2020-eular.4477DOI: 10.1136/annrheumdis-2020-eular.4477
Proinflammatory Neutrophil Function Is Modulated During Secukinumab Therapy in Psoriatic Arthritis Without Compromising Host Defence (Conference Paper)
Moots, R., Cross, A., Wright, H., Edwards, S., Goodson, N., Hawkes, J., . . . Frankland, H. (2020). Proinflammatory Neutrophil Function Is Modulated During Secukinumab Therapy in Psoriatic Arthritis Without Compromising Host Defence. In ARTHRITIS & RHEUMATOLOGY Vol. 72. Retrieved from https://www.webofscience.com/
2019
014 APPA inhibits neutrophil pro-inflammatory functions without impairing host defence: is this a potential new therapy for arthritis? (Journal article)
Cross, A. L., Hawkes, J. J., Wright, H. L., Moots, R. J., & Edwards, S. W. (2019). 014 APPA inhibits neutrophil pro-inflammatory functions without impairing host defence: is this a potential new therapy for arthritis?. Rheumatology, 58(Supplement_3). doi:10.1093/rheumatology/kez106.013DOI: 10.1093/rheumatology/kez106.013
APPA, A POTENTIAL NEW THERAPY FOR OSTEOARTHRITIS, INHIBITS NEUTROPHIL PRO-INFLAMMATORY FUNCTIONS WITHOUT IMPAIRING HOST DEFENCE (Conference Paper)
Cross, A., Hawkes, J., Wright, H., Larkins, N., Edwards, S., & Moots, R. (2019). APPA, A POTENTIAL NEW THERAPY FOR OSTEOARTHRITIS, INHIBITS NEUTROPHIL PRO-INFLAMMATORY FUNCTIONS WITHOUT IMPAIRING HOST DEFENCE. In OSTEOARTHRITIS AND CARTILAGE Vol. 27 (pp. S189-S190). doi:10.1016/j.joca.2019.02.290DOI: 10.1016/j.joca.2019.02.290
APPA INHIBITS NEUTROPHIL PRO-INFLAMMATORY FUNCTIONS WITHOUT IMPAIRING HOST DEFENCE: IS THIS A POTENTIAL NEW THERAPY FOR ARTHRITIS? (Conference Paper)
Cross, A. L., Hawkes, J. J., Wright, H. L., Moots, R. J., & Edwards, S. W. (2019). APPA INHIBITS NEUTROPHIL PRO-INFLAMMATORY FUNCTIONS WITHOUT IMPAIRING HOST DEFENCE: IS THIS A POTENTIAL NEW THERAPY FOR ARTHRITIS?. In RHEUMATOLOGY Vol. 58 (pp. 46). Retrieved from https://www.webofscience.com/
2018
Phoomvuthisarn, P., Cross, A., Glennon-Alty, L., Wright, H. L., & Edwards, S. W. (2018). The CDK inhibitor purvalanol A induces neutrophil apoptosis and increases the turnover rate of Mcl-1: potential role of p38-MAPK in regulation of Mcl-1 turnover. CLINICAL AND EXPERIMENTAL IMMUNOLOGY, 192(2), 171-180. doi:10.1111/cei.13107DOI: 10.1111/cei.13107
250 The effect of JAK inhibition on neutrophil killing, netosis and metabolism in rheumatoid arthritis (Journal article)
Chokesuwattanaskul, S., Mangalakumaran, S., Chapman, E., Cross, A., Phelan, M. M., Lian, L. -Y., . . . Wright, H. L. (2018). 250 The effect of JAK inhibition on neutrophil killing, netosis and metabolism in rheumatoid arthritis. Rheumatology, 57(suppl_3). doi:10.1093/rheumatology/key075.474DOI: 10.1093/rheumatology/key075.474
THE EFFECT OF JAK INHIBITION ON NEUTROPHIL KILLING, NETOSIS AND METABOLISM IN RHEUMATOID ARTHRITIS (Conference Paper)
Chokesuwattanaskul, S., Mangalakumaran, S., Chapman, E., Cross, A., Phelan, M. M., Lian, L. -Y., . . . Wright, H. L. (2018). THE EFFECT OF JAK INHIBITION ON NEUTROPHIL KILLING, NETOSIS AND METABOLISM IN RHEUMATOID ARTHRITIS. In RHEUMATOLOGY Vol. 57. Retrieved from https://www.webofscience.com/
2017
Goebel, A., Lee, M. K., Cacciola, F., Cross, A., & Eldridge, P. (2018). A technique to assess perineuronal mediators. BRITISH JOURNAL OF NEUROSURGERY, 32(6), 697-699. doi:10.1080/02688697.2017.1416058DOI: 10.1080/02688697.2017.1416058
OBSERVATIONAL STUDY ON THE EFFECTS OF IL-6 INHIBITOR THERAPY ON MYOSTATIN IN PATIENTS WITH RHEUMATOID ARTHRITIS (Conference Paper)
Chapman, M. J., Narayanan, R. P., Cross, A., Moots, R., Wilding, J., & Goodson, N. (2017). OBSERVATIONAL STUDY ON THE EFFECTS OF IL-6 INHIBITOR THERAPY ON MYOSTATIN IN PATIENTS WITH RHEUMATOID ARTHRITIS. In ANNALS OF THE RHEUMATIC DISEASES Vol. 76 (pp. 852). doi:10.1136/annrheumdis-2017-eular.5831DOI: 10.1136/annrheumdis-2017-eular.5831
2016
EFFECTS OF APPA ON HUMAN NEUTROPHIL FUNCTION (Journal article)
Cross, A., Hawkes, J., Moots, R., & Nicholas, L. (2016). EFFECTS OF APPA ON HUMAN NEUTROPHIL FUNCTION. OSTEOARTHRITIS AND CARTILAGE, 24, S335-S336. doi:10.1016/j.joca.2016.01.601DOI: 10.1016/j.joca.2016.01.601
2015
Steele, C. A., Powell, J., Kemp, G., Halford, J., Wilding, J., Harrold, J., . . . Daousi, C. (2015). Cerebral activations during viewing of food stimuli in adult patients with acquired structural hypothalamic damage: a functional neuroimaging study. International Journal of Obesity, 39, 1376-1382. doi:10.1038/ijo.2015.82DOI: 10.1038/ijo.2015.82
Serum Adiponectin Determines Circulating Vitamin D Concentration Independently of Other Adipokines (Conference Paper)
Narayanan, R. P., Boyle, L. D., Ng, S., Kahlon, A., Cross, A., Ding, C., . . . Wilding, J. P. H. (2015). Serum Adiponectin Determines Circulating Vitamin D Concentration Independently of Other Adipokines. In DIABETES Vol. 64 (pp. A574). Retrieved from https://www.webofscience.com/
2014
236. Microparticle Micrornas Correlate with Disease Activity in Rheumatoid Arthritis and May Play a Significant Role in Pathogenesis (Journal article)
Bishop, K., Winget, C., Cross, A., Barnes, T., Hawkes, J., Moots, R., . . . Edwards, S. (2014). 236. Microparticle Micrornas Correlate with Disease Activity in Rheumatoid Arthritis and May Play a Significant Role in Pathogenesis. Rheumatology, 53(suppl_1), i148. doi:10.1093/rheumatology/keu117.005DOI: 10.1093/rheumatology/keu117.005
Effects of IL-6 and IL-6 blockade on neutrophil function in vitro and in vivo (Journal article)
Wright, H. L., Cross, A. L., Edwards, S. W., & Moots, R. J. (2014). Effects of IL-6 and IL-6 blockade on neutrophil function in vitro and in vivo. Rheumatology, 53(7), 1321-1331. doi:10.1093/rheumatology/keu035DOI: 10.1093/rheumatology/keu035
MICROPARTICLE microRNAs CORRELATE WITH DISEASE ACTIVITY IN RHEUMATOID ARTHRITIS AND MAY PLAY A SIGNIFICANT ROLE IN PATHOGENESIS (Conference Paper)
Bishop, K., Winget, C., Cross, A., Barnes, T., Hawkes, J., Moots, R., . . . Edwards, S. (2014). MICROPARTICLE microRNAs CORRELATE WITH DISEASE ACTIVITY IN RHEUMATOID ARTHRITIS AND MAY PLAY A SIGNIFICANT ROLE IN PATHOGENESIS. In RHEUMATOLOGY Vol. 53 (pp. 148). Retrieved from https://www.webofscience.com/
2013
Inhibition of pre-B cell colony-enhancing factor (PBEF/NAMPT/visfatin) decreases the ability of human neutrophils to generate reactive oxidants but does not impair bacterial killing (Journal article)
Roberts, K. J., Cross, A., Vasieva, O., Moots, R. J., & Edwards, S. W. (2013). Inhibition of pre-B cell colony-enhancing factor (PBEF/NAMPT/visfatin) decreases the ability of human neutrophils to generate reactive oxidants but does not impair bacterial killing. JOURNAL OF LEUKOCYTE BIOLOGY, 94(3), 481-492. doi:10.1189/jlb.1012527DOI: 10.1189/jlb.1012527
Importance of Brain Reward Circuitry in Adult Patients with Acquired Structural Hypothalamic Damage: a Functional Neuroimaging Study (Journal article)
Steele, C. A., Powell, J. L., Kemp, G. J., Halford, J. C., Wilding, J. P., Harrold, J., . . . Cuthbertson, D. J. (2013). Importance of Brain Reward Circuitry in Adult Patients with Acquired Structural Hypothalamic Damage: a Functional Neuroimaging Study.
2012
Effects of chronic treatment with metformin on dipeptidyl peptidase-4 activity, glucagon-like peptide 1 and ghrelin in obese patients with Type 2 diabetes mellitus (Journal article)
Thondam, S. K., Cross, A., Cuthbertson, D. J., Wilding, J. P., & Daousi, C. (2012). Effects of chronic treatment with metformin on dipeptidyl peptidase-4 activity, glucagon-like peptide 1 and ghrelin in obese patients with Type 2 diabetes mellitus. DIABETIC MEDICINE, 29(8), E205-E210. doi:10.1111/j.1464-5491.2012.03675.xDOI: 10.1111/j.1464-5491.2012.03675.x
2011
Relative α<sub>1</sub>-anti-trypsin deficiency in systemic sclerosis (Journal article)
Barnes, T. C., Cross, A., Anderson, M. E., Edwards, S. W., & Moots, R. J. (2011). Relative α<sub>1</sub>-anti-trypsin deficiency in systemic sclerosis. RHEUMATOLOGY, 50(8), 1373-1378. doi:10.1093/rheumatology/ker123DOI: 10.1093/rheumatology/ker123
2008
The dual effects of TNFα on neutrophil apoptosis are mediated via differential effects on expression of Mcl-1 and Bfl-1 (Journal article)
Cross, A., Moots, R. J., & Edwards, S. W. (2008). The dual effects of TNFα on neutrophil apoptosis are mediated via differential effects on expression of Mcl-1 and Bfl-1. BLOOD, 111(2), 878-884. doi:10.1182/blood-2007-05-087833DOI: 10.1182/blood-2007-05-087833
'Effect of sera from patients with systemic sclerosis and Raynaud's phenomenon on neutrophil apoptosis' (Journal article)
Naylor, E., Barnes, T., Cross, A., Anderson, M., Edwards, S., & Moots, R. (2008). 'Effect of sera from patients with systemic sclerosis and Raynaud's phenomenon on neutrophil apoptosis'. Rheumatology, 47, *.
Neutrophil cytoskeletal abnormalities in systemic sclerosis. (Journal article)
Barnes, T., Cross, A., Naylor, E., Anderson, M., Edwards, S., & Moots, R. (2008). Neutrophil cytoskeletal abnormalities in systemic sclerosis.. Rheumatology, Suppl(47), ii304.
2007
Microbial mannan inhibits bacterial killing by macrophages: A possible pathogenic mechanism for Crohn's disease (Journal article)
Mpofu, C. M., Campbell, B. J., Subramanian, S., Marshall-Clarke, S., Hart, C. A., Cross, A., . . . Rhodes, J. M. (2007). Microbial mannan inhibits bacterial killing by macrophages: A possible pathogenic mechanism for Crohn's disease. GASTROENTEROLOGY, 133(5), 1487-1498. doi:10.1053/j.gastro.2007.08.004DOI: 10.1053/j.gastro.2007.08.004
2006
Neutrophil apoptosis in rheumatoid arthritis is regulated by local oxygen tensions within joints (Journal article)
Cross, A., Barnes, T., Bucknall, R. C., Edwards, S. W., & Moots, R. J. (2006). Neutrophil apoptosis in rheumatoid arthritis is regulated by local oxygen tensions within joints. JOURNAL OF LEUKOCYTE BIOLOGY, 80(3), 521-528. doi:10.1189/jlb.0306178DOI: 10.1189/jlb.0306178
Sodium salicylate promotes neutrophil apoptosis by stimulating caspase-dependent turnover of Mcl-1 (Journal article)
Derouet, M., Thomas, L., Moulding, D. A., Akgul, C., Cross, A., Moots, R. J., & Edwards, S. W. (2006). Sodium salicylate promotes neutrophil apoptosis by stimulating caspase-dependent turnover of Mcl-1. JOURNAL OF IMMUNOLOGY, 176(2), 957-965. doi:10.4049/jimmunol.176.2.957DOI: 10.4049/jimmunol.176.2.957
Induction of cancer cell apoptosis by inhibition of NF-kB and COX-2 mediated expression of Mcl-1 using time lapse imaging of individual living cells (Journal article)
Cross, A., Thomas, L. W., White, M. R. H., Edwards, S. W., & Moots, R. J. (2006). Induction of cancer cell apoptosis by inhibition of NF-kB and COX-2 mediated expression of Mcl-1 using time lapse imaging of individual living cells. North West Cancer Research Fund (Annual Report), 1, 23.
Sodium salicylate promotes neutrophil apoptosis by stimulating caspase-dependent turnover of Mcl-1 (Journal article)
Derouet, M., Thomas, L., Moulding, D. A., Akgul, C., Cross, A., Moots, R. J., & Edwards, S. W. (2006). Sodium salicylate promotes neutrophil apoptosis by stimulating caspase-dependent turnover of Mcl-1. Journal of Immunology, 176(2), 957-965.
2005
Effects of Sodium Salicylate on Neutrophil Apoptosis: Acceleration of Mcl-1 Turnover (Journal article)
Cross, A., Derouet, M., Thomas, L., & Moots, R. J. (2005). Effects of Sodium Salicylate on Neutrophil Apoptosis: Acceleration of Mcl-1 Turnover. Arthritis and Rheumatism, 44, I19.
FUNCTIONAL ANALYSIS OF MCL-1 (Journal article)
Cross, A., Thomas, L., & Edwards, S. (2005). FUNCTIONAL ANALYSIS OF MCL-1. North West Cancer Research Fund (Annual report), 1, 20.
Neutrophil gene expression in rheumatoid arthritis. (Journal article)
Cross, A., Bakstad, D., Allen, J. C., Thomas, L., Moots, R. J., & Edwards, S. W. (2005). Neutrophil gene expression in rheumatoid arthritis.. Pathophysiology : the official journal of the International Society for Pathophysiology, 12(3), 191-202. doi:10.1016/j.pathophys.2005.07.006DOI: 10.1016/j.pathophys.2005.07.006
Regulation of Neutrophil Apoptosis by TNFalpha (Journal article)
Cross, A., Derouet, M., Edwards, S. W., & Moots, R. J. (2005). Regulation of Neutrophil Apoptosis by TNFalpha. Arthritis and Rheumatism, 52(9), 480-481.
The pathogenesis of systemic sclerosis - insights from the innate immune system (Journal article)
Anderson, M. E., Cross, A., Edwards, S. W., Pazmany, L., & Moots, R. J. (2005). The pathogenesis of systemic sclerosis - insights from the innate immune system. Rheumatology, 44, i129.
2004
Granulocyte macrophage colony-stimulating factor signaling and proteasome inhibition delay neutrophil apoptosis by increasing the stability of Mcl-1 (Journal article)
Derouet, M., Thomas, L., Cross, A., Moots, R. J., & Edwards, S. W. (2004). Granulocyte macrophage colony-stimulating factor signaling and proteasome inhibition delay neutrophil apoptosis by increasing the stability of Mcl-1. JOURNAL OF BIOLOGICAL CHEMISTRY, 279(26), 26915-26921. doi:10.1074/jbc.M313875200DOI: 10.1074/jbc.M313875200
Secretion of oncostatin M by neutrophils in rheumatoid arthritis (Journal article)
Cross, A., Edwards, S. W., Bucknall, R. C., & Moots, R. J. (2004). Secretion of oncostatin M by neutrophils in rheumatoid arthritis. ARTHRITIS AND RHEUMATISM, 50(5), 1430-1436. doi:10.1002/art.20166DOI: 10.1002/art.20166
2003
Synovial fluid neutrophils transcribe and express class II major histocompatibility complex molecules in rheumatoid arthritis (Journal article)
Cross, A., Bucknall, R. C., Cassatella, M. A., Edwards, S. W., & Moots, R. J. (2003). Synovial fluid neutrophils transcribe and express class II major histocompatibility complex molecules in rheumatoid arthritis. ARTHRITIS AND RHEUMATISM, 48(10), 2796-2806. doi:10.1002/art.11253DOI: 10.1002/art.11253
Control of Neutrophil Apoptosis in Rheumatoid Arthritis (Journal article)
Cross, A., Moots, R. J., Bucknall, R. C., & Edwards., S. W. (2003). Control of Neutrophil Apoptosis in Rheumatoid Arthritis. Arthritis and Rheumatism, 48(9), S472.
Control of neutrophil apoptosis in rheumatoid arthritis (Journal article)
Cross, A., Moots, R. J., Bucknall, R. C., & Edwards, S. W. (2003). Control of neutrophil apoptosis in rheumatoid arthritis. ARTHRITIS AND RHEUMATISM, 48(9), S472.
2002
Control of Neutrophil Apoptosis in Rheumatoid Arthritis: Expression of Bax and Mcl-1 (Journal article)
Cross, A., Moots, R. J., Bucknall, R., & Edwards, S. W. (2002). Control of Neutrophil Apoptosis in Rheumatoid Arthritis: Expression of Bax and Mcl-1. Arthritis and Rheumatism, 46(9), S421.
Control of neutrophil apoptosis in rheumatoid arthritis: Expression of Bax and Mcl-1 (Journal article)
Cross, A., Moots, R. J., Bucknall, R., & Edwards, S. W. (2002). Control of neutrophil apoptosis in rheumatoid arthritis: Expression of Bax and Mcl-1. ARTHRITIS AND RHEUMATISM, 46(9), S421.
Secretion of Oncostatin M by Neutrophils in Rheumatoid Arthritis (Journal article)
Cross, A., Moots, R. J., Bucknall, R., & Edwards, S. W. (2002). Secretion of Oncostatin M by Neutrophils in Rheumatoid Arthritis. Arthritis and Rheumatism, 46(9), S562.
Secretion of oncostatin M by neutrophils in rheumatoid arthritis (Journal article)
Cross, A., Moots, R. J., Bucknall, R., & Edwards, S. W. (2002). Secretion of oncostatin M by neutrophils in rheumatoid arthritis. ARTHRITIS AND RHEUMATISM, 46(9), S562.
Undated
Sustained T Cell Immunity, Protection and Boosting Using Extended Dosing Intervals of BNT162b2 mRNA Vaccine (Journal article)
Payne, R. P., Longet, S., Austin, J. A., Skelly, D., Dejnirattisai, W., Adele, S., . . . Consortium, T. P. I. T. C. H. (n.d.). Sustained T Cell Immunity, Protection and Boosting Using Extended Dosing Intervals of BNT162b2 mRNA Vaccine.
Angyal, A., Longet, S., Moore, S., Payne, R. P., Harding, A., Tipton, T., . . . Group, P. I. T. C. H. C. (n.d.). T-Cell and Antibody Responses to First BNT162b2 Vaccine Dose in Previously SARS-CoV-2-Infected and Infection-Naive UK Healthcare Workers: A Multicentre, Prospective, Observational Cohort Study.